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Post-Concussion Dizziness

Symptoms persisting after a concussion, such as headaches and dizziness.

Post-Concussion Dizziness: How Physiotherapy Can Restore Your Balance and Clear the Fog

Three weeks ago, you hit your head during a soccer game. The initial symptoms—headache, confusion, nausea—have mostly faded. But the dizziness? It won't go away.

You feel unsteady when you walk. Turning your head makes the room spin. Busy environments like grocery stores leave you disoriented and exhausted. You can't focus at work because looking at your computer screen triggers waves of dizziness. Your doctor said to "give it time," but you're wondering: Is this normal? Will it ever get better?

Post-concussion dizziness is one of the most common and debilitating persistent symptoms following a mild traumatic brain injury, affecting up to 80% of concussion patients and significantly impacting daily function, return to work, and return to sports [Farì et al., 2024; Art et al., 2023; Ellis, Leddy & Willer, 2015].

Here's the encouraging news: Post-concussion dizziness is highly treatable with specialized vestibular rehabilitation. Research demonstrates that multimodal physiotherapy—combining vestibular exercises, cervical spine treatment, and aerobic conditioning—can dramatically accelerate recovery, with 73% of patients medically cleared for return to activity within 8 weeks compared to only 7% receiving standard care [Schneider et al., 2014; Farì et al., 2024; Art et al., 2023].

At Vaughan Physiotherapy Clinic, we specialize in evidence-based rehabilitation for post-concussion dizziness. We understand that your symptoms aren't "all in your head"—they're the result of specific, treatable dysfunctions in your vestibular system, cervical spine, and autonomic regulation that require targeted intervention, not just time.

What Is Post-Concussion Dizziness?

Understanding the Problem

Post-concussion dizziness refers to persistent vestibular symptoms—dizziness, vertigo, imbalance, and disequilibrium—that continue beyond the typical 7-10 day recovery window following a concussion or mild traumatic brain injury (mTBI) [Farì et al., 2024; Ellis, Leddy & Willer, 2015].

Prevalence:

  • Dizziness is one of the most common symptoms following concussion [Farì et al., 2024; Art et al., 2023]
  • Can affect up to 80% of concussion patients at some point during recovery
  • When symptoms persist beyond 3 weeks, it meets criteria for Post-Concussion Syndrome (PCS) [Ellis, Leddy & Willer, 2015]
  • Approximately 15-20% of concussion patients develop persistent symptoms requiring specialized intervention [Ellis, Leddy & Willer, 2015]

Types of Post-Concussion Dizziness

Post-concussion dizziness isn't a single condition—it represents distinct operational Post-Concussion Disorders (PCDs) that require different treatment approaches [Ellis, Leddy & Willer, 2015].

1. Vestibulo-Ocular PCD [Ellis, Leddy & Willer, 2015]

What It Is:Persistent symptoms caused by dysfunction of the vestibular and oculomotor systems—damage to the balance organs in your inner ear and the systems that coordinate eye movements.

Characteristic Symptoms:

  • Dizziness and light-headedness
  • Gait instability and balance problems
  • Visual disturbances like blurred or double vision
  • Motion sensitivity
  • Difficulty with visual tracking
  • Symptoms worsen with head movement

The Mechanism:The concussion has disrupted how your vestibular system (inner ear balance organs) communicates with your visual system. Your brain is receiving conflicting information about head position and movement, resulting in dizziness and imbalance.

Key Feature:Problems with balance, spatial orientation, and visual processing during movement.

2. Cervicogenic PCD [Ellis, Leddy & Willer, 2015]

What It Is:Persistent symptoms caused by dysfunction of the cervical spine somatosensory system—damage to the neck structures and nerves that provide sensory information about head and neck position.

Characteristic Symptoms:

  • Neck pain and stiffness
  • Occipital headaches (pain at the base of the skull)
  • Postural imbalance
  • Dizziness that seems to originate from the neck
  • Reduced neck range of motion
  • Tenderness in neck muscles

The Mechanism:The same forces that caused your concussion often cause whiplash-type cervical injuries. Damage to neck joints, muscles, and proprioceptors (position sensors) disrupts the sensory feedback your brain needs for balance and spatial orientation.

Key Feature:Neck-related symptoms with dizziness that worsens with neck movement or certain head positions.

3. Visual Vertigo Component

What It Is:Many post-concussion patients develop visual motion sensitivity—dizziness triggered by busy visual environments or moving visual stimuli [Bronstein et al., 2013; Thompson et al., 2015].

Characteristic Symptoms:

  • Dizziness in supermarkets, malls, or crowded spaces
  • Symptoms triggered by watching action movies or scrolling screens
  • Disorientation in visually complex environments
  • Worsening with fluorescent lights or visual patterns
  • Feeling overwhelmed by moving traffic

The Mechanism:After concussion, your brain may develop an overreliance on visual input for balance (visual dependency), leading to dizziness when visual information is complex or ambiguous [Bronstein et al., 2013].

4. PPPD (Persistent Postural-Perceptual Dizziness)

What It Is:A functional disorder of chronic dizziness where the brain's balance control system becomes "stuck" in a maladaptive pattern following the initial concussion [Thompson et al., 2015].

Characteristic Symptoms:

  • Persistent, non-spinning dizziness or unsteadiness (>3 months)
  • Symptoms exacerbated by:
    • Upright posture
    • Your own motion (head/body movement)
    • Complex visual stimuli
    • Objects moving in the environment

The Mechanism:The initial protective response to concussion (stiffened gait, increased reliance on vision, heightened vigilance) fails to resolve. The nervous system remains stuck in "high alert" mode, creating persistent symptoms even after the brain has healed [Thompson et al., 2015].

Trigger:Concussion/mild traumatic brain injury is a common trigger for PPPD development [Thompson et al., 2015].

Recognizing Post-Concussion Dizziness: Key Symptoms

Primary Vestibular Symptoms

Dizziness [Farì et al., 2024; Art et al., 2023]:

  • Feeling of lightheadedness
  • Sensation of being "off-balance"
  • Non-specific feeling of disorientation

Vertigo [Art et al., 2023]:

  • Spinning sensation
  • Feeling like the room is moving
  • Can be constant or episodic
  • Often triggered by specific head positions or movements

Imbalance and Unsteadiness [Ellis, Leddy & Willer, 2015]:

  • Difficulty walking straight
  • Feeling like you might fall
  • Need to hold onto walls or furniture
  • Particularly problematic in the dark or on uneven surfaces

Disequilibrium [Ellis, Leddy & Willer, 2015]:

  • General sense of spatial disorientation
  • Difficulty with coordination
  • Trouble navigating complex environments

Associated Symptoms

Visual Disturbances [Ellis, Leddy & Willer, 2015]:

  • Blurred vision
  • Double vision (diplopia)
  • Difficulty focusing
  • Visual tracking problems
  • Sensitivity to light (photophobia)
  • Symptoms worse with screen time or reading

Neck-Related Symptoms [Farì et al., 2024; Ellis, Leddy & Willer, 2015]:

  • Neck pain and stiffness
  • Occipital headaches
  • Limited neck range of motion
  • Pain that radiates from neck to head

Cognitive Effects [Ellis, Leddy & Willer, 2015]:

  • Difficulty concentrating (especially with dizziness)
  • Mental fogginess or "brain fog"
  • Feeling overwhelmed in busy environments
  • Slowed processing speed

Autonomic Symptoms:

  • Nausea (especially with vertigo)
  • Fatigue (disproportionate to activity)
  • Sensitivity to noise (phonophobia)
  • Sweating or pallor with severe dizziness

Exacerbating Factors

Post-concussion dizziness is typically worsened by specific activities and environments:

Movement-Related:

  • Quick head movements (turning to look, nodding)
  • Bending over or looking up
  • Rolling over in bed
  • Getting up from lying or sitting
  • Walking, especially quickly
  • Running or athletic movements

Visual Triggers:

  • Busy visual environments (stores, malls)
  • Screen time (computers, phones, TV)
  • Reading
  • Scrolling or moving visual patterns
  • Fluorescent or flickering lights
  • Watching fast-paced action scenes

Cognitive Demands:

  • Multitasking
  • Concentrating for extended periods
  • Complex problem-solving while moving
  • Navigating unfamiliar environments

Environmental:

  • Crowded or noisy spaces
  • Dim lighting or darkness
  • Uneven surfaces
  • Moving vehicles (as passenger)

The Impact on Daily Life

Post-concussion dizziness can be profoundly disabling:

Work/School:

  • Difficulty concentrating at desk/computer
  • Problems navigating hallways and classrooms
  • Inability to attend long meetings or lectures
  • Reduced productivity and performance

Social Activities:

  • Avoiding restaurants, movies, social gatherings
  • Isolation due to symptom unpredictability
  • Anxiety about leaving home
  • Difficulty participating in conversations in noisy environments

Physical Activities:

  • Unable to exercise or play sports
  • Fear of falling or re-injury
  • Difficulty with previously simple tasks (grocery shopping, cooking)
  • Reduced independence

Psychological Impact:

  • Anxiety about symptoms and recovery
  • Depression related to functional limitations
  • Fear of permanent disability
  • Frustration with "invisible" symptoms

What Causes Post-Concussion Dizziness?

The Biomechanical Injury

The Initial Trauma [Farì et al., 2024; Art et al., 2023]:A concussion is induced by direct or indirect biomechanical forces that cause the brain to move forward and backward inside the skull.

Dual Injury Pattern:The same forces typically cause:

  1. Brain injury (concussion/mTBI)
  2. Cervical injury (whiplash-type forces to neck)

This explains why many post-concussion patients have both vestibular and cervicogenic contributions to their dizziness.

The Neurometabolic Cascade

What Happens to Your Brain [Ellis, Leddy & Willer, 2015]:

Concussion triggers a cascade of molecular processes:

  • Alterations in cell membrane permeability
  • Disrupted ion transport
  • Abnormal neurotransmitter release
  • Changes in cellular metabolism
  • Disrupted cerebral blood flow (CBF)

The "Acute Energy Crisis":Following initial hypermetabolism, the brain enters a protracted period of decreased glucose utilization and metabolism [Ellis, Leddy & Willer, 2015].

Why This Causes Dizziness:The vestibular system and its central processing pathways are metabolically demanding. During the energy crisis, these systems may not function optimally, resulting in:

  • Impaired vestibular signal processing
  • Delayed or inaccurate balance responses
  • Difficulty integrating multiple sensory inputs
  • Symptoms that worsen with any additional metabolic demand

Specific Vestibular System Damage

Direct Vestibular Injury:The biomechanical forces can cause:

  • Inner ear damage (labyrinthine concussion)
  • Otolith displacement or damage
  • Semicircular canal dysfunction
  • Vestibular nerve injury

Result:Asymmetric vestibular input to the brain, causing vertigo, imbalance, and spatial disorientation.

Cervical Spine Contribution

Whiplash-Type Forces [Ellis, Leddy & Willer, 2015]:The same acceleration-deceleration forces affecting the brain also injure cervical structures:

Damaged Structures:

  • Facet joints (neck joints)
  • Intervertebral discs
  • Neck muscles (strains, spasms)
  • Cervical proprioceptors (position sensors)

Why Neck Injury Causes Dizziness:The cervical spine contains dense proprioceptive input that your brain uses for:

  • Determining head position in space
  • Coordinating eye-head-neck movements
  • Maintaining balance and posture

When these proprioceptors are damaged or sending inaccurate signals, your brain receives conflicting information about head position, resulting in dizziness and imbalance—this is cervicogenic dizziness.

Visual System Disruption

Oculomotor Dysfunction [Ellis, Leddy & Willer, 2015]:Concussion can disrupt:

  • Eye tracking (smooth pursuit)
  • Eye jumps (saccades)
  • Vestibulo-ocular reflex (VOR) (keeping vision stable during head movement)
  • Convergence (focusing on near objects)

Result:Blurred vision, difficulty reading, visual-vestibular mismatch leading to dizziness.

Autonomic Dysregulation

Disrupted Autonomic Control:Concussion can affect the autonomic nervous system's regulation of:

  • Cerebral blood flow
  • Heart rate response to position changes
  • Blood pressure regulation

Result:Orthostatic intolerance (dizziness when standing), exercise intolerance, symptoms that worsen with exertion.

The Maladaptive Response (PPPD Development)

The Protective Pattern [Thompson et al., 2015]:Initially after concussion, your nervous system adopts protective strategies:

  • Stiffened gait ("high-risk postural control")
  • Increased reliance on visual input
  • Heightened vigilance about balance
  • Reduced movement to avoid symptoms

When It Becomes Problematic:In some patients, this protective pattern fails to resolve even after the brain has healed. The nervous system remains stuck in "high alert" mode—this is PPPD [Thompson et al., 2015].

Contributing Factors to Chronicity:

  • Anxiety about symptoms and re-injury
  • Avoidance behaviors (limiting movement to prevent dizziness)
  • Catastrophic thinking about recovery
  • Lack of appropriate early rehabilitation

Risk Factors for Persistent Dizziness

Pre-Injury Risk Factors

History of Previous Concussions [Ellis, Leddy & Willer, 2015]:

  • Prior concussion is a risk factor for prolonged recovery
  • Each subsequent concussion may take longer to resolve
  • Cumulative effects on vestibular system

Female Gender [Ellis, Leddy & Willer, 2015; Art et al., 2023]:

  • Females have higher risk of prolonged recovery following concussion
  • Female athletes have higher incidence of concussion than males
  • Hormonal influences on recovery may play a role

Younger Age [Ellis, Leddy & Willer, 2015; Art et al., 2023]:

  • Adolescent athletes at higher risk of concussion than adults
  • May require longer recovery period
  • Developing brains may be more vulnerable

Pre-Existing Conditions [Ellis, Leddy & Willer, 2015]:

  • History of migraine headaches (strong risk factor for prolonged recovery)
  • Anxiety or depression
  • Motion sickness susceptibility
  • Previous vestibular disorders

Injury-Related Risk Factors

Severity of Initial Symptoms:

  • More severe acute symptoms
  • Loss of consciousness
  • Post-traumatic amnesia
  • Prolonged confusion

Presence of Multiple Symptom Types:

  • Combined vestibular, cervicogenic, and cognitive symptoms
  • Visual disturbances
  • Severe headaches

Post-Injury Risk Factors

Inadequate Early Management [Ellis, Leddy & Willer, 2015; Farì et al., 2024]:

Returning to Activity Too Soon:

  • Premature return to sports, work, or school
  • Risk of symptom exacerbation
  • Prolonged recovery timeline
  • Risk of second-impact syndrome (in acute phase)

Excessive Rest:

  • Excessive rest is associated with slower recovery [Farì et al., 2024]
  • Leads to deconditioning
  • Increases anxiety and avoidance behaviors
  • May perpetuate symptoms

Psychological Factors:

  • Development of fear-avoidance behaviors
  • Catastrophic thinking about recovery
  • Anxiety sensitization to symptoms
  • Social isolation

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Why Physiotherapy Is Essential for Post-Concussion Dizziness

The Evidence: Multimodal Rehabilitation Works

Landmark Research [Schneider et al., 2014; Farì et al., 2024; Art et al., 2023]:

A randomized controlled trial comparing standard care to combined cervical spine and vestibular rehabilitation found:

Dramatic Results:

  • 73% of combined intervention group medically cleared for return to sport within 8 weeks
  • Only 7% of standard care control group achieved clearance in same timeframe
  • Patients in treatment group were 10.27 times more likely to be medically cleared within 8 weeks

What This Means:Active, targeted physiotherapy intervention produces outcomes more than 10 times better than passive "rest and wait" approach.

Why Multimodal Treatment Is Superior

Addressing Multiple Dysfunctions:Post-concussion dizziness rarely has a single cause. Most patients have overlapping dysfunctions requiring comprehensive treatment [Ellis, Leddy & Willer, 2015]:

  1. Vestibular dysfunction (inner ear/balance system)
  2. Cervical spine dysfunction (neck injury contributing to dizziness)
  3. Visual-vestibular mismatch (eye movement problems)
  4. Autonomic dysregulation (blood flow/heart rate issues)
  5. Maladaptive movement patterns (fear-avoidance, protective posturing)

Single-Modality Limitations:Treating only one component leaves other dysfunctions unaddressed, limiting recovery.

The Core Components That Work

1. Vestibular Rehabilitation Therapy (VRT) [Ellis, Leddy & Willer, 2015; Farì et al., 2024; Murray et al., 2017]

Purpose:Improve functional recovery outcomes such as gaze stabilization, balance, gait, and return to work/sport.

Mechanism:VRT aims to re-calibrate depth and spatial perception by re-establishing efficient integration of vestibular, visual, and somatosensory systems [Ellis, Leddy & Willer, 2015].

Key Exercises:

Gaze Stabilization (VOR Training):

  • Improves vestibulo-ocular reflex
  • Stabilizes vision during head movement
  • Reduces dizziness with movement

Balance Training:

  • Progressive challenges from stable to unstable surfaces
  • Eyes open to eyes closed progression
  • Static to dynamic activities
  • Reduces fall risk, improves confidence

Habituation Exercises:

  • Repeated exposure to movements that provoke dizziness
  • Gradual desensitization
  • Brain learns to filter and adapt to provocative stimuli

Evidence:VRT improves functional recovery outcomes and is essential for vestibulo-ocular PCD [Murray et al., 2017; Farì et al., 2024].

2. Cervical Spine Treatment [Schneider et al., 2014; Ellis, Leddy & Willer, 2015; Art et al., 2023]

Purpose:Restore normal somatosensory output from cervical afferents (neck position sensors).

Interventions:

  • Manual therapy of cervical and thoracic spine
  • Head-neck proprioception re-training
  • Therapeutic exercises
  • Postural correction
  • Soft tissue techniques

Often Includes:Balance and gaze stabilization exercises as cervical proprioception integrates with vestibular function [Ellis, Leddy & Willer, 2015].

Evidence:Combined cervical and vestibular treatment more effective than either alone [Schneider et al., 2014; Farì et al., 2024].

3. Sub-Symptom Threshold Aerobic Exercise (SSTAE) [Farì et al., 2024; Art et al., 2023; Ellis, Leddy & Willer, 2015]

Purpose:Promote recovery of physiologic PCD—persistent symptoms from altered brain metabolism.

Mechanism:

  • Improves autonomic nervous system (ANS) regulation
  • Increases cerebral blood flow (CBF)
  • Normalizes brain metabolism
  • Prevents deconditioning

Protocol:

  • Start below symptom threshold
  • Gradually increase intensity and duration
  • Monitor heart rate (initially 60-80%, progress to 70-85% max HR)
  • Frequency: 3-5 times per week

Evidence:

  • Safe and effective in reducing symptom intensity and improving cognitive function [Langevin et al., 2020; Farì et al., 2024]
  • Accelerates recovery: Median 13-14 days vs. 17-19 days for control groups [Leddy et al., 2019, 2021; Art et al., 2023]

4. Visual-Vestibular Integration Training

Purpose:Address visual motion sensitivity and visual-vestibular mismatch.

Techniques:

  • Optokinetic stimulation
  • Visual desensitization exercises (e.g., spinning umbrella)
  • Complex environment exposure (grocery store protocol)
  • VOR exercises with varied visual backgrounds

Evidence:Effective for post-concussion patients with visual vertigo component [Bronstein et al., 2013; Thompson et al., 2015].

5. Cognitive-Behavioral Strategies [Thompson et al., 2015]

Purpose:Address maladaptive patterns, anxiety, and fear-avoidance that perpetuate symptoms.

Interventions:

  • Education about symptom mechanisms
  • Graded exposure to feared activities
  • Anxiety management techniques
  • Challenging catastrophic thoughts
  • Promoting active coping strategies

Evidence:Particularly important for patients developing PPPD following concussion [Thompson et al., 2015].

The Comprehensive Advantage

Synergistic Effects:When combined, these interventions address:

  • ✓ Inner ear dysfunction
  • ✓ Neck-related dizziness
  • ✓ Visual processing problems
  • ✓ Brain metabolic recovery
  • ✓ Autonomic regulation
  • ✓ Psychological barriers
  • ✓ Deconditioning

Result:Faster, more complete recovery than any single intervention alone.

Recovery Timeline: What to Expect with Physiotherapy

Without Specialized Treatment

Natural History:

  • Most concussions (80-85%) resolve within 1-2 weeks [Ellis, Leddy & Willer, 2015]
  • 15-20% develop persistent symptoms (Post-Concussion Syndrome) [Ellis, Leddy & Willer, 2015]
  • Persistent dizziness can last months to years without appropriate intervention
  • Standard care (rest only): Only 7% cleared for return to activity within 8 weeks [Schneider et al., 2014]

With Multimodal Physiotherapy

Short-Term Improvement (6-12 Weeks)

Combined Intervention Outcomes [Schneider et al., 2014; Farì et al., 2024; Art et al., 2023]:

  • 73% medically cleared within 8 weeks (vs. 7% with standard care)
  • 10.27 times more likely to achieve medical clearance
  • Significant improvement in:
    • Dizziness severity
    • Balance confidence
    • Functional abilities
    • Return to activities

Symptom Reduction [Grabowski et al., 2017]:

  • Statistically significant symptom reduction
  • Post-Concussion Symptom Scale scores: 18.2 to 9.1 (approximately 50% reduction)

Aerobic Exercise Benefits [Leddy et al., 2019, 2021; Art et al., 2023]:

  • Faster recovery: Median 13-14 days (aerobic group) vs. 17-19 days (control)
  • Approximately 25-30% reduction in recovery time

Functional Milestones

Weeks 1-2:

  • Reduction in symptom severity at rest
  • Improved tolerance for basic activities
  • Ability to perform gentle exercises without exacerbation

Weeks 3-6:

  • Significant improvement in balance confidence
  • Increased tolerance for visual stimuli
  • Progressive return to work/school activities
  • Improvement in neck pain and headaches

Weeks 6-12:

  • Substantial functional improvement
  • Many patients achieve medical clearance
  • Return to most or all pre-injury activities
  • Continued refinement of balance and coordination

Individual Variation

Factors Affecting Timeline:

  • Severity of initial injury
  • Number and type of dysfunctions present
  • Adherence to rehabilitation program
  • Presence of complicating factors (previous concussions, comorbidities)
  • Timing of treatment initiation (earlier = better)

Realistic Expectations:

  • Some patients recover within weeks
  • Others require 2-3 months of intensive therapy
  • A small percentage need longer-term management
  • Early intervention produces best outcomes

Long-Term Prognosis

With Appropriate Treatment:

  • Most patients achieve full or near-full recovery
  • Return to work, school, sports, and recreational activities
  • Restored confidence in movement and balance
  • Resolution of anxiety about symptoms

Importance of Complete Recovery:Achieving full recovery before returning to contact sports is essential to prevent:

  • Cumulative effects of multiple concussions
  • Increased risk of prolonged symptoms with future injuries
  • Long-term cognitive and balance problems

Your Personalized Treatment Plan

Initial Comprehensive Assessment

We evaluate all potential contributors:

Vestibular System Testing:

  • Gaze stability (VOR function)
  • Dynamic visual acuity
  • Balance testing (modified Clinical Test of Sensory Interaction on Balance)
  • Positional testing
  • Head impulse testing

Cervical Spine Examination:

  • Range of motion
  • Joint mobility
  • Muscle tenderness and spasm
  • Proprioceptive testing
  • Trigger point identification

Oculomotor Assessment:

  • Smooth pursuit
  • Saccades
  • Convergence
  • Near-point convergence

Functional Testing:

  • Gait analysis
  • Dynamic Gait Index
  • Balance confidence scales
  • Activities-specific Balance Confidence scale
  • Dizziness Handicap Inventory

Exertional Testing:

  • Heart rate and symptom response to graded exercise
  • Determination of symptom threshold
  • Assessment of autonomic regulation

Phase 1: Foundation and Stabilization (Weeks 1-3)

Cervical Spine Treatment

Manual Therapy:

  • Joint mobilization for restricted segments
  • Soft tissue release for muscle tension
  • Gentle traction as needed

Therapeutic Exercise:

  • Deep neck flexor strengthening
  • Postural correction exercises
  • Cervical proprioceptive retraining
  • Range of motion restoration

Goal:Restore normal neck function and eliminate cervicogenic contribution to dizziness.

Basic Vestibular Exercises

Gaze Stabilization (VOR x1):

  • Focus on stationary target while moving head
  • Start seated, progress to standing
  • Horizontal and vertical planes
  • 1-2 minutes, 2-3 times daily

Static Balance:

  • Romberg stance (feet together)
  • Tandem stance (heel-to-toe)
  • Single-leg stance
  • Eyes open, then eyes closed progression
  • Each position: 30-60 seconds, repeat 3 times

Sub-Symptom Threshold Aerobic Exercise

Starting Protocol:

  • Light walking or stationary cycling
  • Heart rate: 60-70% of maximum
  • Duration: 10-20 minutes
  • Frequency: Daily or every other day
  • Critical rule: Stop if symptoms increase beyond mild

Monitoring:

  • Track symptoms before, during, and after
  • Gradually increase duration and intensity
  • Goal: Build tolerance without triggering setbacks

Phase 2: Progressive Challenge (Weeks 4-8)

Advanced Vestibular Training

VOR x2 Exercises:

  • Target moves opposite direction to head
  • Increases challenge to vestibular system
  • Improves dynamic visual stability

Habituation Exercises:

  • Repeated exposure to movements that provoke mild dizziness
  • Head rotations at increasing speeds
  • Looking up/down repeatedly
  • Quick position changes
  • Goal: Desensitization through controlled exposure

Dynamic Balance:

  • Walking with head turns
  • Tandem gait (heel-to-toe walking)
  • Walking while reading
  • Obstacle navigation
  • Foam surface challenges
  • Eyes closed variations

Visual-Vestibular Integration

Optokinetic Exercises:

  • Spinning umbrella technique
  • Complex visual environment exposure
  • Screen-based visual motion tolerance
  • Busy environment training (graduated exposure)

Real-World Practice:

  • Grocery store protocol (gradually increasing duration)
  • Crowded environment exposure
  • Driving simulation (as passenger first)

Intensified Aerobic Training

Progression:

  • Heart rate: 70-85% of maximum
  • Duration: 20-40 minutes
  • Frequency: 4-5 times per week
  • Incorporate sport-specific movements as tolerated

Phase 3: Return-to-Activity (Weeks 8-12+)

Sport-Specific Training

Progressive Stages:

  1. Light aerobic activity (achieved in Phase 2)
  2. Sport-specific exercise (no contact)
  3. Non-contact training drills
  4. Full-contact practice
  5. Return to play

Criteria for Advancement:

  • No symptom exacerbation at current level
  • Minimum 24 hours between stages
  • Medical clearance before full return

Work/School Reintegration

Graduated Return-to-Learn:

  • Progressive increase in cognitive demands
  • Extended breaks initially
  • Accommodations as needed:
    • Reduced workload
    • Extended time on tasks
    • Preferential seating
    • Modified schedule
  • Full reintegration as symptoms allow

Functional Independence

Real-World Activities:

  • Full participation in daily activities
  • Driving
  • Social engagements
  • Recreational activities
  • Exercise and fitness routines

Ongoing Monitoring and Adjustment

Regular Reassessment:

  • Objective measures (balance testing, VOR gains)
  • Subjective scales (DHI, ABC)
  • Functional performance
  • Symptom tracking

Program Modifications:

  • Based on progress and response
  • Addressing new challenges as they arise
  • Ensuring continued advancement

Prevention Strategies

Primary Prevention: Avoiding Initial Injury

Sports Safety:

  • Proper equipment (helmets, mouthguards)
  • Rule enforcement (reducing dangerous play)
  • Proper technique training
  • Limiting contact in practice
  • Baseline concussion testing for athletes

General Safety:

  • Seatbelt use
  • Fall prevention (home safety, proper footwear)
  • Workplace safety protocols

Secondary Prevention: Avoiding Persistent Symptoms

Immediate Post-Concussion:

  • Proper acute management (initial rest, early evaluation)
  • Avoiding premature return to activity
  • Following graduated return protocols
  • Avoiding second impact during acute phase [Ellis, Leddy & Willer, 2015]

Early Intervention:

  • Seek specialized care if symptoms persist beyond 7-10 days
  • Don't wait weeks or months hoping symptoms will resolve
  • Early physiotherapy prevents chronic patterns
  • Address cervical spine injury promptly

Appropriate Activity Levels:

  • Balance between rest and activity
  • Avoid excessive rest (associated with slower recovery) [Farì et al., 2024]
  • Follow SSTAE protocols under professional guidance
  • Gradual, systematic progression

Psychological Support:

  • Address anxiety about symptoms early
  • Prevent development of fear-avoidance behaviors
  • Education about expected recovery
  • Cognitive-behavioral strategies if needed

Tertiary Prevention: Avoiding Recurrence

Complete Recovery Before Return:

  • Full medical clearance required
  • Graduated return-to-play protocol completed
  • No symptoms at maximum exertion
  • Objective measures normalized

Long-Term Strategies:

  • Strengthening neck musculature
  • Maintaining cardiovascular fitness
  • Balance and coordination training
  • Awareness of cumulative concussion effects

Informed Decision-Making:

  • Understanding risks of multiple concussions
  • Considering retirement from high-risk sports after multiple injuries
  • Lifetime perspective on brain health

Frequently Asked Questions

"Why is my dizziness lasting so long?"

Post-concussion dizziness persists because:

Multiple Systems Affected:Your dizziness likely has overlapping causes [Ellis, Leddy & Willer, 2015]:

  • Vestibular system dysfunction (inner ear)
  • Cervical spine injury (neck)
  • Visual processing problems
  • Autonomic dysregulation
  • Maladaptive nervous system patterns

Energy Crisis:Your brain is in a protracted metabolic recovery phase with decreased glucose utilization [Ellis, Leddy & Willer, 2015]. The metabolically-demanding vestibular system struggles to function optimally.

Incomplete Compensation:Without targeted rehabilitation, your brain may not fully compensate for damaged systems, leaving you with persistent symptoms.

Good News:Persistent dizziness does NOT mean permanent damage. With appropriate multimodal treatment, 73% of patients achieve medical clearance within 8 weeks [Schneider et al., 2014].

"Will my dizziness ever go away?"

Yes—with appropriate treatment, most patients achieve significant improvement or complete resolution.

The Evidence:

  • 73% cleared for return to activity within 8 weeks with combined cervical/vestibular rehab [Schneider et al., 2014]
  • Symptom scores can reduce by 50% (from 18.2 to 9.1) with targeted intervention [Grabowski et al., 2017]
  • Recovery time reduced by 25-30% with aerobic exercise protocols [Leddy et al., 2019, 2021]

Important:

  • Early intervention produces better outcomes
  • "Wait and see" approach often leads to prolonged symptoms
  • Active rehabilitation superior to passive rest

"Can I exercise if I'm still dizzy?"

Yes—strategic, symptom-limited exercise is not only safe but beneficial [Farì et al., 2024; Art et al., 2023].

Sub-Symptom Threshold Aerobic Exercise (SSTAE):

  • Start below your symptom threshold
  • Light walking or stationary cycling
  • Monitor symptoms carefully
  • Stop if symptoms increase beyond mild
  • Gradually progress as tolerance improves

The Evidence:

  • Safe and effective in reducing symptoms [Langevin et al., 2020; Farì et al., 2024]
  • Accelerates recovery compared to rest alone [Leddy et al., 2019, 2021]
  • Improves cerebral blood flow and metabolic recovery [Ellis, Leddy & Willer, 2015]

What to Avoid:

  • High-intensity exercise that significantly worsens symptoms
  • Contact sports until fully cleared
  • Activities with high fall risk during acute symptomatic phase

Professional Guidance Essential:Work with a physiotherapist to determine appropriate:

  • Starting intensity
  • Progression rate
  • Exercise selection
  • Warning signs to stop

"Do I need medication?"

Medication is sometimes helpful but not always necessary [Ellis, Leddy & Willer, 2015; Thompson et al., 2015].

When Medication May Help:

  • Migraine prophylaxis (if headaches predominate)
  • SSRIs/SNRIs (for PPPD, anxiety, or depression)
  • Vestibular suppressants (SHORT-term only for severe acute vertigo)
  • Sleep aids (if insomnia is preventing recovery)

Important Considerations:

  • Physiotherapy is first-line treatment for post-concussion dizziness
  • Many patients recover with rehab alone
  • Medications work best combined with active rehabilitation
  • Avoid long-term vestibular suppressants (may inhibit compensation)

Collaborative Approach:Your physiotherapist and physician should coordinate care, with medication supporting—not replacing—active rehabilitation.

"How long before I can return to sports/work?"

Timeline varies but is dramatically improved with specialized treatment:

With Multimodal Physiotherapy [Schneider et al., 2014]:

  • 73% medically cleared within 8 weeks
  • Return often possible in 6-12 weeks with appropriate progression

Without Specialized Treatment:

  • Only 7% cleared within 8 weeks
  • May take months to a year
  • Some never fully recover

Factors Affecting Timeline:

  • Severity and complexity of symptoms
  • Number of systems affected (vestibular, cervical, visual)
  • Adherence to rehabilitation
  • Previous concussion history
  • Age and general health

Critical Requirements for Return:

  • Symptom-free at rest AND with exertion
  • Normal objective testing
  • Completion of graduated return protocol
  • Medical clearance
  • No symptom recurrence with sport-specific activities

Don't Rush:Premature return risks:

  • Prolonged recovery
  • Worsening symptoms
  • Re-injury with potentially worse outcomes

Why Choose Vaughan Physiotherapy Clinic for Post-Concussion Dizziness?

Post-concussion dizziness is frustrating and frightening. When weeks turn into months and you're still dizzy, it's natural to worry that you'll never feel normal again. At Vaughan Physiotherapy Clinic, we want you to know: persistent dizziness after concussion is highly treatable, and you don't have to accept it as your new normal.

We specialize in evidence-based, multimodal rehabilitation that addresses ALL the potential causes of your dizziness—not just one piece of the puzzle. Our approach is grounded in the research showing that combined interventions produce outcomes more than 10 times better than standard care alone.

What sets us apart:

  • Comprehensive assessment identifying vestibular, cervical, visual, and autonomic contributions
  • Evidence-based protocols proven to accelerate recovery
  • Multimodal treatment approach addressing all dysfunctions simultaneously
  • Individualized exercise prescription based on your specific deficits and symptom threshold
  • Graduated return-to-activity protocols ensuring safe, timely clearance
  • Coordination with physicians for comprehensive concussion management
  • Education and support throughout your recovery journey

Most importantly, we understand the impact post-concussion dizziness has on every aspect of your life. We're committed to helping you return to work, school, sports, and daily activities as quickly and safely as possible—with confidence that your symptoms are truly resolved, not just masked.

Ready to Overcome Post-Concussion Dizziness?

Our Specialized Multimodal Approach to Post-Concussion Rehabilitation

Our comprehensive programs include:

  • Combined vestibular and cervical spine rehabilitation proven to produce 10x better outcomes
  • Gaze stabilization exercises (VOR training) for visual-vestibular integration
  • Balance retraining progressed from basic to sport-specific challenges
  • Cervical spine manual therapy and therapeutic exercise
  • Sub-symptom threshold aerobic exercise (SSTAE) protocols
  • Visual desensitization for visual motion sensitivity
  • Habituation exercises for movement-provoked dizziness
  • Cognitive-behavioral strategies for anxiety and fear-avoidance
  • Graduated Return-to-Play and Return-to-Learn protocols
  • Collaboration with physicians, neurologists, and sports medicine specialists

Book Your Assessment Today:

📞 Phone: 905-669-1221

📍 Location: 398 Steeles Ave W #201, Thornhill, ON L4J 6X3

🌐 Online Booking: www.vaughanphysiotherapy.com

Don't let post-concussion dizziness keep you sidelined. The research is clear: specialized multimodal physiotherapy produces dramatically better outcomes than "wait and see" approaches. With 73% of patients achieving medical clearance within 8 weeks using combined interventions—compared to only 7% with standard care—the evidence speaks for itself. Your dizziness is treatable. Contact us today to start your evidence-based recovery journey.

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