Gluteal tendinopathy is the most common cause of lateral hip pain, affecting the tendons of the gluteus medius and minimus. Evidence-based physiotherapy with progressive exercise produces superior long-term outcomes compared to corticosteroid injections or wait-and-see approaches.
Gluteal tendinopathy is a degenerative overuse condition affecting the tendons of the gluteus medius and gluteus minimus muscles where they insert onto the greater trochanter of the femur. It is the most common cause of lateral hip pain and was historically referred to as "trochanteric bursitis," although research now confirms that the primary pathology lies in the tendons rather than the bursa (Gill et al., 2025, Cureus). Like other tendinopathies, it represents a failed healing response within the tendon characterized by disorganized collagen, increased ground substance, and neovascularization — a process called tendinosis rather than active inflammation.
Gluteal tendinopathy affects approximately 10–25% of the general population and is particularly prevalent among:
The condition has been described as having a comparable impact on quality of life to severe hip osteoarthritis, with significant effects on sleep, mobility, and participation in daily activities.
The gluteal tendons are the primary stabilizers of the pelvis during single-leg activities — which accounts for approximately 85% of the gait cycle.
The gluteus medius is a fan-shaped muscle that originates from the outer surface of the ilium and inserts onto the lateral and superoposterior facets of the greater trochanter. It has three functional segments: anterior fibers (hip flexion and internal rotation), middle fibers (primary hip abduction), and posterior fibers (hip extension and external rotation). The tendon has a layered, complex insertion similar to the rotator cuff of the shoulder, earning the gluteal tendons the nickname "rotator cuff of the hip."
The gluteus minimus lies deep to the gluteus medius, originating from the lower portion of the ilium and inserting onto the anterior facet of the greater trochanter. It works as a hip abductor and stabilizer, and its anterior fibers are important for controlling femoral rotation during gait.
The greater trochanter is the bony prominence on the outer upper femur where both gluteal tendons attach. In gluteal tendinopathy, the tendons become compressed against the greater trochanter, particularly during positions of hip adduction. This compressive load is a key driver of pain and pathology (Grimaldi et al., 2025, Archives of Orthopaedic and Trauma Surgery).
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The evidence overwhelmingly supports physiotherapy-led exercise as the first-line treatment for gluteal tendinopathy. The landmark LEAP trial (Mellor et al., 2018, BMJ) — a randomized clinical trial of 204 participants — compared education plus exercise, corticosteroid injection, and a "wait and see" approach. At eight weeks, the education-plus-exercise group achieved significantly better outcomes than both other groups. At one year, the exercise group maintained superior outcomes, while the corticosteroid group showed no lasting benefit.
Patricio Cordeiro et al. (2024, Scientific Reports) confirmed that structured exercise programs produce significant improvements in pain and function. Wang et al. (2025, Journal of Orthopaedic Surgery and Research) found that exercise-based approaches ranked highest for both short- and long-term pain reduction.
Corticosteroid injections provide short-term relief (4–6 weeks) but the LEAP trial demonstrated they offer no benefit beyond one year and may impair long-term tendon healing (Mellor et al., 2018, BMJ).
Is gluteal tendinopathy the same as trochanteric bursitis?
Not exactly. Research shows the primary pathology is in the gluteal tendons, not the bursa (Gill et al., 2025, Cureus). The more accurate term is gluteal tendinopathy or greater trochanteric pain syndrome.
Should I get a cortisone injection?
The LEAP trial showed injections offer no benefit beyond one year compared to doing nothing (Mellor et al., 2018, BMJ). Exercise therapy produced superior outcomes at both 8 weeks and 12 months.
Can I still exercise?
Yes — the key is modifying exercise selection to avoid compressive activities while progressively loading the tendon under physiotherapy guidance.
Why does my hip hurt more at night?
Lying on the affected side compresses the gluteal tendons against the greater trochanter. Sleep on your back or unaffected side with a pillow between your knees.
Is stretching good for gluteal tendinopathy?
Stretches that pull the leg across the body compress the tendon and can worsen the condition. Focus on strengthening exercises instead.
How long until I can return to running?
Most patients begin a graduated walk-run program between 8–16 weeks, provided they can perform single-leg exercises pain-free.
Do I need imaging?
Usually no. Gluteal tendinopathy is a clinical diagnosis. Grimaldi et al. (2025, Archives of Orthopaedic and Trauma Surgery) found imaging findings did not reliably predict treatment outcomes.
At Vaughan Physiotherapy, our experienced team uses the latest evidence-based approaches to help you overcome gluteal tendinopathy. Book your appointment today:
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